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The latest research proposes the key mechanism of thyroid homeostasis disorder caused by bacterial LPS, including increased expression of Tg and NIS gene, TLR4-B (NF-kB) pathway and regulating the activities of different deiodinase (type I and type II) [107]

The honest answer is that the evidence cannot say, because no study has ever compared them directly in the same model, let alone in humans

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