The dynamics of BPC are not well understood, including where it goes, how it works, and its effects, such as the results of 80mg enemas of BPC for colitis, although phase one and two trials showed no adverse effects and a positive signal for ulcer colitis, respectively 1m4s
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Given that the pathogenesis of antiretroviral toxic neuropathy is based on the disruption of mitochondrial energy supply one can assume that the clinical effect of ALCAR is dependent on its mitochondrial availability
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