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Animals were randomly divided into three experimental groups and inoculated with either the wt D39 strain, the gpoA mutant, or the isogenic revertant gpoA + strain

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This is why combining both pathways (as in CagriSema) produces greater weight loss than either alone

Research shows that in senescent cells, p53 levels are maintained low through ubiquitination while its pro-apoptotic activity is repressed through nuclear segregation by FOXO4. The peptide's competitive inhibition restores p53 activity by promoting nuclear exclusion, allowing p53 to execute its pro-apoptotic program and eliminate senescent cells through targeted pathway activation

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