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Immune checkpoint molecules like PD-1 (programmed death-1) and CTLA-4 (Cytotoxic T lymphocyte-associated antigen-4) function as negative regulators of immune activation, and when triggered by tumor or tumor-associated cells, they suppress immune responses (105, 125128)

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Acetyl coenzyme A (Acetyl-CoA), produced through the metabolism of glucose, fatty acids, and amino acids [7], is utilized in cholesterol synthesis, a process regulated by SREBP-2 through the enzyme 3-hydroxy-3-methylglutaryl-CoA reductase (HMGCR), the rate-limiting enzyme in cholesterol biosynthesis