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A role for genetic has been demonstrated, with evidence of an increased risk of NAFL in patients with polymorphisms, like those associated to patatin-like phospholipase domain containing 3 (PNPLA3) rs738409, transmembrane 6 superfamily member 2 (TM6SF2) rs58542926 and membrane bound O-acyltransferase domain containing 7 (MBOAT7) rs641738 (4447)

Some studies involving healthy subjects reported that endogenous GIP contributes to up to 25% of the suppression of bone resorption after a meal, while it found that endogenous GLP-1 has no impact on postprandial bone homeostasis [53, 54]

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Analytical Biochemistry, 307(1), 7683

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